03477naa a2200157 a 450000100080000000500110000800800410001910000110006024501040007126000090017552028690018465300250305365300260307865300220310477301930312610862762012-07-27       2011    bl uuuu     u00u1 u    #d1 aEpagri  aDevelopment of CO2 injury in 'Fuji' apples treated with 1-MCP and stored in controlled atmospheres.  c2011  aThe development of internal CO2 injury symptoms in apple fruit treated and untreated with 1-MCP was studied for ?Fuji? and its clones ?Fuji Suprema? and ?Kiku? stored in CA with 1.6 KPa O2. The evolution of internal CO2 injury symptoms (brown-heart and cavities) throughout a six month storage period was evaluated for fruit from three orchards and stored in CA with high (2.5 kPa) CO2. The timing of high (2.5 kPa) CO2 exposure effects on CO2 injury development were also assessed for late harvested fruit from three orchards stored in CA with low (&lt0.6 kPa) CO2 for 24 weeks, high (2.5 kPa) CO2 for 24 weeks or high (2.5 kPa) CO2 for a single 3 week period (1st to 3rd, 4th to 6th, 7th to 9th or 10th to 12th weeks) plus low (&lt0.6 kPa) CO2 for 21 weeks. The CA delay effects on development of CO2 injury were evaluated for fruit from 83 orchards stored in air at 0 oC for 0 to 4 weeks before storage in CA with 1 kPa CO2. Incidence of fruit with light brownheart injury increased significantly from 30 to 60 days of storage and then decreased from 60 to 120 days while incidence of fruit with moderate and severe brownheart injury increased significantly from 45 to 60 days storage, regardless of 1-MCP treatment, when stored in 2.5 kPa CO2. Incidence of fruit with one cavity increased from 60 to 120 days while incidence of fruit with 2 or more cavities increased after 120 days storage. 1-MCP treatment did not affect the incidence of fruit with cavities or light and moderate brownheart symptoms, regardless of storage period. The incidence of fruit with severe brownheart was higher in 1-MCP treated fruit only after 150 and 180 days of storage. Brownheart and cavity incidence were highest when fruit were exposed to 2.5 kPa CO2 during the first 3 weeks of CA storage. Sensitivity to CO2 injuries decreased continually from the first to the tenth week in storage similarly for untreated and 1-MCP-treated fruit. The result indicated that the period of maximum sensitivity to 2.5 kPa CO2 was not clearly altered by 1-MCP treatment. The delay of CA (1.6 kPa O2 + 1 kPa CO2) reduced the incidence of CO2 injury for both untreated and 1-MCP-treated fruit. However, the incidence of fruit with CO2 injury in 1 kPa CO2 CA was higher in fruit treated with 1-MCP compared with untreated controls, particularly when CA was delayed for 0 to 3 weeks. Results indicated that a 2 to 3 weeks CA delay is required for 1-MCP treated fruits to attain susceptibility to CO2 injury similar to that of untreated controls stored in rapid CA (0 week delay). The development of CO2 injury was significantly lower in ?Fuji Suprema? fruit than that in ?Fuji? and ?Fuji Kiku?, regardless of CA delay period. There was significant correlation between fruit CO2 injury and firmness, acidity, starch index and nitrogen content at harvest, although the correlation indexes were low.  a1-methylcyclopropene  aControlled atmosphere  aMalus x domestica  tIn: FOURTH INTERNACIONAL CONFERE POSTHARVEST UNLIMITED, 4., 2011, Leavenworth. [Proceedings...]. Leavenworth: ISHS International Society for Horticultural Science Postharvest, 2011. p. 36.